Definition/General

Introduction:
-Apocrine carcinoma of the breast is a rare subtype of invasive ductal carcinoma characterized by cells with apocrine features, such as abundant eosinophilic granular cytoplasm and prominent nucleoli
-It accounts for about 1-4% of all breast cancers.
Origin:
-It is thought to arise from the terminal duct-lobular unit (TDLU) and shows differentiation towards apocrine-type cells, similar to those found in apocrine sweat glands.
Classification:
-It is classified as a special subtype of invasive ductal carcinoma
-The diagnosis requires that at least 90% of the tumor cells show apocrine morphology
-It can be in situ (apocrine DCIS) or invasive.
Epidemiology:
-It tends to occur in older women, with a peak incidence in the 6th and 7th decades
-The clinical presentation and risk factors are generally similar to those of invasive ductal carcinoma of no special type.

Clinical Features

Presentation:
-Usually presents as a palpable breast mass
-Nipple discharge is uncommon
-It can be detected by mammography, often as an irregular mass with or without calcifications.
Symptoms:
-A firm, often painless breast lump is the most common symptom
-Skin changes or nipple retraction are less frequent unless the tumor is large or centrally located.
Risk Factors:
-General risk factors for breast cancer apply, such as age, family history, and hormonal factors
-There are no specific risk factors uniquely associated with apocrine carcinoma.
Screening:
-Mammography and ultrasound are the primary imaging modalities
-The imaging features are often indistinguishable from common types of invasive ductal carcinoma.

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Gross Description

Appearance:
-The gross appearance is not specific and resembles that of invasive ductal carcinoma, no special type
-It is typically a firm, gray-white mass with irregular or spiculated margins.
Characteristics:
-The tumor can range in size
-Necrosis may be present in larger tumors
-The consistency is usually firm to hard.
Size Location:
-Can occur anywhere in the breast, with no specific predilection for any quadrant
-Size is variable at diagnosis.
Multifocality:
-Multifocality and multicentricity can occur, similar to other types of invasive ductal carcinoma.

Microscopic Description

Histological Features:
-The tumor is composed of sheets, nests, or glands of large polygonal cells with abundant, granular, eosinophilic cytoplasm
-The cell borders are distinct
-Apical snouts or decapitation secretions are characteristic features.
Cellular Characteristics:
-The nuclei are typically large, round to oval, and often have prominent, centrally located eosinophilic nucleoli
-Nuclear pleomorphism can be variable
-Mitotic figures are usually present.
Architectural Patterns:
-The growth pattern can be solid, glandular, or papillary
-A desmoplastic stromal response is common.
Grading Criteria:
-Grading is done using the Nottingham grading system, similar to other invasive breast carcinomas
-Most apocrine carcinomas are high-grade (Grade 2 or 3).

Immunohistochemistry

Positive Markers:
-Characteristically positive for Androgen Receptor (AR)
-Also positive for GCDFP-15 (Gross Cystic Disease Fluid Protein-15)
-Most are negative for Estrogen Receptor (ER) and Progesterone Receptor (PR)
-HER2 overexpression is common (about 30-50% of cases).
Negative Markers:
-Usually negative for ER and PR
-A significant portion are HER2 negative as well, making them triple-negative (but AR positive).
Diagnostic Utility:
-IHC is crucial for diagnosis
-AR and GCDFP-15 positivity confirms apocrine differentiation
-ER, PR, and HER2 status are vital for determining treatment options.
Molecular Subtypes:
-Most apocrine carcinomas fall into the molecular apocrine or luminal B (HER2-positive) subtypes
-A subset are triple-negative but AR positive.

Molecular/Genetic

Genetic Mutations:
-PIK3CA and TP53 mutations are common
-There is a characteristic pattern of chromosomal gains and losses, including gains of 1q and losses of 1p, 16q, and 22q.
Molecular Markers:
-High expression of androgen-regulated genes
-The molecular profile is distinct from other breast cancer subtypes.
Prognostic Significance:
-The prognosis is generally similar to or slightly better than that of grade-matched invasive ductal carcinoma, no special type
-Prognosis is largely dependent on tumor size, grade, lymph node status, and HER2 status.
Therapeutic Targets:
-HER2-positive cases are treated with HER2-targeted therapies (e.g., trastuzumab)
-For AR-positive, ER/PR-negative tumors, anti-androgen therapy is a potential treatment strategy under investigation.

Differential Diagnosis

Similar Entities:
-Invasive ductal carcinoma with apocrine metaplasia
-Oncocytic carcinoma
-Granular cell tumor
-Lipid-rich carcinoma.
Distinguishing Features:
-Apocrine metaplasia is benign and lacks the malignant nuclear features
-Oncocytic carcinomas have similar eosinophilic cytoplasm but are typically AR negative
-Granular cell tumors are S100 positive
-Lipid-rich carcinomas have clear, vacuolated cytoplasm.
Diagnostic Challenges:
-The main challenge is distinguishing pure apocrine carcinoma from invasive ductal carcinoma with focal apocrine features
-The 90% cutoff for apocrine morphology is used for diagnosis of pure apocrine carcinoma.
Rare Variants:
-Apocrine DCIS is the in-situ counterpart
-Invasive apocrine carcinoma can have various patterns, including papillary and solid.

Sample Pathology Report

Template Format

Sample Pathology Report

Complete Report: This is an example of how the final pathology report should be structured for this condition.

Specimen Information

[specimen type], measuring [size] cm in greatest dimension

Diagnosis

[diagnosis name]

Classification

Classification: [classification system] [grade/type]

Histological Features

Shows [architectural pattern] with [nuclear features] and [mitotic activity]

Size and Extent

Size: [X] cm, extent: [local/regional/metastatic]

Margins

Margins are [involved/uninvolved] with closest margin [X] mm

Lymphovascular Invasion

Lymphovascular invasion: [present/absent]

Lymph Node Status

Lymph nodes: [X] positive out of [X] examined

Special Studies

IHC: [marker]: [result]

Molecular: [test]: [result]

[other study]: [result]

Final Diagnosis

Final diagnosis: [complete diagnosis]